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Norswertianolin acts as a CSE activator and is isolated from G. acuta. Norswertianolin may be a potential agent for cardiovascular diseases.

Norswertianolin (3.125~50 μM; H9c2 cells) has the protective effects on H₂O₂-induced H9c2 cells. Norswertianolin significantly increases the transcription levels of HO-1 and GCLC in H9c2 cells. Norswertianolin (0~150 μM; HepG2 cells) high concentration increases the CSE protein level.

Norswertianolin (4.4 mg/kg/day; s.c.; 1 week) improves CSE/H₂S production.
Norswertianolin treatment attenuates the renal tubular injury in acute I/R. Norswertianolin also reduces I/R-stimulated ROS by DHE fluorescence intensity. Norswertianolin has anti-hypertension effects, inhibits vascular remodeling and reduces inflammation effects.

Room temperature in continental US; may vary elsewhere.

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*In solvent : -80°C, 6 months; -20°C, 1 month (protect from light)