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FSL-1 TFA, a bacterial-derived toll-like receptor 2/6 (TLR2/6) agonist, enhances resistance to experimental HSV-2 infection. FSL-1 TFA induces MMP-9 production through TLR2 and NF-κB/AP-1 signaling pathways in monocytic THP-1 cells.

FSL-1 significantly reduces HSV-2 replication in human vaginal epithelial cells (EC).
FSL-1 induces significant resistance to experimental genital HSV-2 infection through elaboration of a specific cytokine response profile.
FSL-1 (50 ng/mL, 24 hours) induces MMP-9 expression at both mRNA and protein levels in human monocytic THP-1 cells.
FSL-1 activates the MAP kinase/NF-κB signaling pathway.

FSL-1 application significantly protectes against genital HSV-2 challenge in mice.

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